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However, the generic antidepressant may be formulated with different non-active ingredients — the stuff that binds and coats the pills – which can affect the rate at which a drug is released. The information contained herein is not intended to cover all possible uses directions, precautions, warnings, drug interactions, allergic reactions, or adverse effects. This makes fluoxetine highly effective in treatment of clinical depression cases where symptoms like depressed mood and lack of energy exist. Fluoxetine affects neurotransmitters, the chemicals that nerves within the brain use to communicate with each other. The serotonin either travels across the (space >= nerves AND space <= attaches)to receptors on the surface of nearby nerves or it attaches to receptors on the surface of the nerve that produced it, to be taken up by the nerve and released again (a process referred to as re-uptake). Fluoxetine works by preventing the reuptake of one neurotransmitter, serotonin, by nerve cells after it has been released. Some patients may experience withdrawal reactions upon stopping fluoxetine.
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The dose of fluoxetine should be gradually reduced when therapy is discontinued. Be sure your doctor knows about all other medicines you use. Skip the missed dose if it is almost time for your next scheduled dose. However, if it is almost time for the next regularly scheduled weekly dose, skip the missed dose and take the next one as directed. Call your doctor for medical advice about side effects.
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You should consult with a medical professional if you have any questions about your health. Years of development and testing finally led to approval of fluoxetine for marketing. Three randomized, double-blind, placebo-controlled studies showed a decrease in the frequency and severity of migraine headaches with fluoxetine therapy. Because uptake inactivates serotonin by removing it from the synaptic cleft, uptake inhibition by fluoxetine enhances serotonergic function. Fluoxetine does not interact directly with postsynaptic serotonin receptors, muscarinic-cholinergic receptors, histaminergic receptors, or alpha-adrenergic receptors. The liver then metabolizes fluoxetine into norfluoxetine, a desmethyl metabolite, which is also a serotonin reuptake inhibitor. Fluoxetine versus other types of pharmacotherapy for depression.
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Possible involvement of cholinergic and opioid receptor mechanisms in fluoxetine mediated antinociception response in streptozotocin-induced diabetic mice. Plasma catecholamine levels after fluoxetine treatment in depressive patients. Fluoxetine for migraine prophylaxis: a double-blind trial. Fluoxetine prophylaxis of migraine. A randomized, double-blind crossover trial of fluoxetine and amitriptyline in the treatment of fibromyalgia. The fetal safety of fluoxetine: a systematic review and meta-analysis. Fluoxetine attenuates alcohol intake and desire to drink.
Fluoxetine monotherapy in attention-deficit/hyperactivity disorder and comorbid non-bipolar mood disorders in children and adolescents. Double-blind trial of fluoxetine: chronic daily headache and migraine. Fluoxetine and premature ejaculation: a double-blind, crossover, placebo-controlled study. Double-blind placebo-controlled trial of fluoxetine in smoking cessation treatment including nicotine patch and cognitive-behavioral group therapy.
The effect of fluoxetine in patients with pain and constipation-predominant irritable bowel syndrome: a double-blind randomized-controlled study. Fluoxetine and fluvoxamine for treatment of chronic pain. Fluoxetine for the treatment of childhood anxiety disorders. The effects of fluoxetine in the overdose patient. Benign course in a child with a massive fluoxetine overdose.
Timing of onset of antidepressant response with fluoxetine treatment. Antidepressants and the risk of suicide, attempted suicide, and overall mortality in a nationwide cohort. Use of fluoxetine in anorexia nervosa before and after weight restoration. These studies indicate that fluoxetine may help to treat depression and obsessive-compulsive disorder in children.
Blackwell’s five-minute veterinary consult clinical companion. Fluoxetine works by affecting a part of your brain that controls your mood. Always speak with your healthcare provider about possible interactions with all prescription drugs, vitamins, herbs and supplements, and over-the-counter drugs that you are taking. Visit your doctor or health care professional for regular checks on your progress.
Fluoxetine is used to treat depression, obsessive-compulsive disorders, panic disorder, and bulimia (binge eating and purging). Fluoxetine affects chemicals in the brain that may become unbalanced and cause depression or mood disturbances, eating disorders, or obsessive or compulsive symptoms. While you are taking fluoxetine you may need to be monitored for worsening symptoms of depression and suicidal thoughts at the start of therapy or when doses are changed. This medication will cause you to become drowsy. Each entry lists the prescription drugs side effects.
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Neuro-transmitters are made and produced by nerve fibres and after that move and affix to close by nerve fibres. Serotonin is certainly one neuro-transmitter that may be introduced by nerve fibres inside the human brain. As uptake is an decisive process for eliminating introduced neuro-transmitters and eliminating all their activities on nearby nerve fibres, the decreased uptake brought on by prozac raises free serotonin compound that may induces nerve units inside the human brain. Fluoxetine dosage will change for different people. Regrettably, anxiety will become even worse after some time as these chemical substances turn into exhausted even more. Most health conditions and drugs may cause the signs of major depression, including unhappiness. Mature people, or anyone that requires numerous prescription drugs daily, have reached exposure to possible medication interactions which usually trigger the signs of major depression.
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Fluoxetine should be offered to a child or young person with moderate to severe major depressive disorder only in combination with psychological therapy. Additionally, only limited information concerning the long-term safety of fluoxetine on growth, puberty, mental, emotional and behavioural development in this age group is available. The dose should be increased carefully to ensure that you receive the lowest effective dose. This is usual because an improvement in depressive symptoms may not occur until after the first few weeks. Abnormal liver function has been reported rarely, with very rare cases of hepatitis. In addition, do not start fluoxetine in a patient who is being treated with linezolid or intravenous methylene blue due to risk of serotonin syndrome.
Consideration should be given to changing the therapeutic regimen, including possibly discontinuing the medication, in patients whose depression is persistently worse, or who are experiencing emergent suicidality or symptoms that might be precursors to worsening depression or suicidality, especially if these symptoms are severe, abrupt in onset, or were not part of the patient’s presenting symptoms. Treatment with fluoxetine and any concomitant serotonergic agents should be discontinued immediately if the above reactions occur, and supportive symptomatic treatment should be initiated. Anaphylactoid reactions, including bronchospasm, angioedema, laryngospasm, and urticaria alone and in combination, have been reported. Fluoxetine should be introduced with care in patients with a history of seizures.